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Patients with New York Heart Association functional class III-IV heart failure, recent stroke or transient ischemic attack in the last six months, acute coronary syndromes in the preceding two months, uncontrolled hypertension, planned coronary revascularization during the study period, prior exposure to ranolazine, or dialysis were excluded from the study population.

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Patients were randomly split into seven dosing groups, including a placebo group.

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Ranolazine and dronedarone induce prolongation of effective refractory period (ERP) and development of post-repolarization refractoriness in atria (PRR, the difference between ERP and APD 70 in atria and between ERP and APD 90 in ventricles; ERP corresponds to APD 70-75 in atria and to APD 90 in ventricles).

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